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Is Obesity Accelerated Aging? A Physiologic Perspective
Explore this recorded session from the American Diabetes Association’s 2026 Scientific Sessions on obesity and accelerated physiological aging, featuring research on how aging reshapes white and brown adipose tissue, adipocyte development, and thermogenic function. The session also examines liver senescence in metabolic disease, including its links to inflammation, fibrosis, and ferroptotic stress, and discusses potential therapeutic approaches.
Overview
Explore this recorded session from the American Diabetes Association’s 2026 Scientific Sessions on obesity and accelerated physiological aging, featuring research on how aging reshapes white and brown adipose tissue, adipocyte development, and thermogenic function. The session also examines liver senescence in metabolic disease, including its links to inflammation, fibrosis, and ferroptotic stress, and discusses potential therapeutic approaches.
Learning Objectives
  • Identify the primary functions of white and brown adipocytes.
  • Assess how age-specific adipocyte progenitor cells contribute to midlife visceral fat expansion.
  • Examine the proposed IL-1β pathway that reduces UCP1 expression in brown adipocytes.
  • Interpret how hepatocyte senescence-signature levels relate to MASH severity.
Faculty
Qiong Annabel Wang, PhD
Kuo Du, PhD
Summary
Availability:
On-Demand
Expires on Jan 01, 2028
Cost:
FREE
Credit Offered:
No Credit Offered
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